Détail du congrès:
Congrès: Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) (9-13 mai 2005)
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Documents disponibles provenant de ce congrès (411)
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Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Mroue G ; Prigent H ; Orlikowski D ; Bohic M ; Baconnier P ; Lofaso F ; Benchetrit G | 2005Communication n° 662 Patients with advanced neuromuscular diseases often require chronic mechanical ventilation. Long term-assisted ventilation often has to address the problem of leakage, whether the device used is a nasal mask or tracheostomy.[...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Riederer I ; Negroni E ; Bencze M ; Savino W ; Mouly V ; Silva-Barbosa SD ; Butler-Browne GS | 2005Communication n° 501 Myoblast transplant therapy (MTT) can be envisioned as a clinical alternative in the treatment of several myopathies. The problems that remain to be solved to improve the efficiency of MTT is the massive early death of trans[...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Riederer I ; Negroni El ; Savino W ; Bencze M ; Mouly V ; Silva-Barbosa SD ; Butler-Browne GS | 2005Communication n° 495 Myoblast transplant therapy (MTT) can be envisioned as a clinical alternative in the treatment of several myopathies. The problems that remain to be solved to improve the efficiency of MTT is the massive early death of trans[...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; de la Porte S ; Voisin V ; Sébrié C ; Matecki S ; Yu H ; Gillet B ; Ramonatxo M ; Israel M | 2005Communication n° 403 A possible treatment for Duchenne muscular dystrophies would be to compensate for dystrophin loss by increasing the expression of utrophin, another cytoskeletal protein of the muscle membrane. We previously found that L-argi[...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Salah N ; Millet G ; André-Schmutz I ; Desforges B ; Olaso R ; Roblot N ; Courageot S ; Bensimon G ; Cavazzana-Calvo M ; Melki J | 2005Communication n° 174 INTRODUCTION : Several studies have demonstrated that adult bone marrow-derived cells (BMDC) can participate in skeletal muscle regeneration after bone marrow transplantation (BMT). However, the mechanisms and identities of [...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Gherardi RK ; Chrétien F ; Caramelle P ; Christov C ; Dreyfus P | 2005Communication n° 321 We have previously developed a murine model of bone marrow (BM) transplantation from B6-TgGFP transgenic mice to normal irradiated B6 mice, the cytoplasmic green fluorescent protein (GFP) being used as an unambiguous marker [...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Lafreniere JF ; Mills PL ; Skuk D ; Tremblay J | 2005Communication n° 302 Introduction: One problem for the clinical application of myoblast transplantation for Duchenne Muscular Dystrophy (DMD) treatment is the poor migration of transplanted cells. Recently, we described a new approach that consi[...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Courdier-Fruh I ; Magyar JP ; Briguet A | 2005Communication n° 27 Previous studies in mdx mice indicate that upregulation of the cytoskeletal protein utrophin, a structural and functional homologue of dystrophin, offers a potential treatment strategy for Duchenne muscular dystrophy (DMD). C[...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Skuk D ; Goulet M ; Roy B ; Tremblay J | 2005Communication n° 139 Introduction : Our Myoblast Allotransplantation Studies In Monkeys Showed That The Absence Of Immunosuppression Leads To Graft Rejection In A Few Days, While A Good Immunosuppression Allows Graft Survival Apparently As Long [...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Avril-Delplanque A ; Rameau P ; Danos O ; Garcia L ; Dreyfus P | 2005Communication n° 184 Adult bone marrow-derived cells (BMDC) are shown to contribute to skeletal muscle regeneration. Following lethal irradiation and GFP expressing bone marrow cells transplantation, GFP+ mononucleated cells are detected among s[...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Lafreniere JF ; Mills PL ; Tremblay J | 2005Communication n° 301 Introduction : Transplantation of myogenic cells is the only treatment that could reconstitute the satellite cell population of the muscle and introduce normal genes in pre-existing myofibers. One major problem that contribu[...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Alameddine HS ; Morgan J ; Bausero P ; Gross J | 2005Communication n° 377 A soluble and membrane-bound metallo-endopeptidase activity has been shown to be involved in myoblast fusion (Couch and Stritmatter 1983, and 1984). Furthermore, MMP-2 and MMP-9 activation and regulation during skeletal musc[...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Charland L ; Lavoie J ; Puymirat J | 2005Communication n° 119 Use of databases (DB) for managing neuromuscular diseases (NMD) data is an impressive tool for physicians and researchers. We have developed a DB on an Oracle platform. Oracle has a solid architecture witch is only limited b[...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Orcesi S ; Rossi M ; Gorni K ; Berardinelli A ; Beghi E ; Angelini C ; Bertini E ; Pini A ; Palmucci L ; Mercuri E ; Lanzi G | 2005Communication n° 196 Introduction : One of the major goals both in the general management of Neuromuscular Disorders (NMDs) and in therapeutic trials is increasingly recognized as the improvement or at least prevention of worsening of heath-rela[...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; El Fahime E ; Benabdallah BF ; Tremblay J | 2005Communication n° 169 Therapies are currently investigating the use of the newly identified pluripotent muscle derived stem cells (MDSCs), as a possible cell-mediated therapy, in order to repopulate the dystrophic muscles. The existence of MDSCs,[...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Marti I ; Demeer B ; Angeard N ; Ben Yaou R ; Daoud F ; Saillour Y ; Récan D ; Heron D ; Rivier F ; Desguerre I ; Tuffery S ; Cossee M ; Leturcq F ; Cuisset JM ; Kaplan JC ; Chelly J | 2005Communication n° 527 Duchenne and Becker muscular dystrophinopathies (DMD and BMD) are allelic X-linked diseases characterized by progressive muscular degeneration, responsible for muscle weakness, and associated with cognitive impairment in app[...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Martinez Perea de Posadas M ; Multicenter Study S ; Many Hospital,Multicenter S | 2005Communication n° 90 INTRODUCTION : About 0.2 % of the disabled population suffer from an inherited Neuromsucular Disorders (NMD). There are no current data about Neuromuscular Diseases Incidence in our country.Each entity presents relative frequ[...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Davoust J ; Ginhoux F ; Turbant S ; LeBoeuf MB ; Gross D ; Lemonnier F ; Goyenvalle A ; Garcia L ; Danos O | 2005Communication n° 668 Cellular immune responses may compromise long-term expression in Dystrophin-based gene therapy treatments. To predict cytotoxic T-cell responses mediated by dystrophin (DYST), we designed a new H-2-negative HLA-A*0201 transg[...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Skuk D ; Goulet M ; Roy B ; Piette V ; Côte C ; Hogrel JY ; Bouchard JP ; Lachance JG ; Sylvain M ; Tremblay J | 2005Communication n°2 Introduction : Following a similar protocol as we used in monkeys, we observed that myoblast transplantation (MT), tested in only 1cm3 of muscle, systematically restored the expression of normal dystrophin in myofibers of DMD p[...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Dorchies OM ; Wagner S ; Waldhauser K ; Vuadens O ; Buetler TM ; Kucera P | 2005Communication n° 435 Duchenne Muscular Dystrophy (DMD) is a frequent inherited muscular disorder which is still fatal. DMD patients present with muscular weakness that progress towards paralysis. They usually die in the third decade because of r[...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Fraysse B ; Liantonio A ; Giannuzzi V ; Camerino C ; Cocchi D ; Conte Camerino D | 2005Communication n° 306 Growth hormone secretagogues (GHS) stimulate GH release through the activation of a pituitary receptor. GHS binding sites were also found in various tissues including skeletal muscle. We recently showed that in vitro applica[...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Briguet A ; Courdier-Fruh I ; Foster M ; Erb M ; Meier T ; Lescop C ; Siendt H ; Herzner H ; Henneboehle M ; Weyermann P ; von Sprecher A ; Magyar J | 2005Communication n° 24 Calpain I and II are ubiquitous calcium-dependent cytosolic cystein proteases that are activated in dystrophin-deficient muscles. Inhibition of calpains by overexpression of its endogenous inhibitor calpastatin has been shown[...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Angeard N ; Gargiulo M ; Jacquette A ; Eymard B ; Heron D | 2005Communication n° 525 Myotonic dystrophy type 1 (DM1) is an autosomal-dominant neuromuscular disease with an incidence of 1 in 8000 individuals. The gene is located on the chromosome 19q13.3 and it encodes for a protein with serine-threonine kina[...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Skuk D ; Roy B ; Goulet M ; Bouchard JP ; Chapdelaine P ; Roy R ; Dugré FJ ; Lachance JG ; Sylvain M | 2005Communication n° 1 Introduction : Used appropriately, myoblast transplantation (MT) could be a potential therapeutic tool in the treatment of myopathies: it can induce the expression of donor proteins in the myofibers of the recipient, and it ca[...]Article
Congrès international de myologie 2005 (International Congress of Myology 2005; 9-13 mai 2005; Nantes, France) ; Boldrin L ; Malerba A ; Flaibani M ; Piccoli M ; Slanzi E ; Pozzobon M ; Messina C ; Zanesco L ; Gamba PG ; Elvassore N ; Vitiello L | 2005Communication n° 565 Introduction : Tissue engineering is a developing strategy to replace or repair congenital or acquired large muscle defects and to improve the outcome of muscle dystrophies and others genetic deficiencies. Objectives : In ou[...]